By Alan H. B. Wu (auth.), Alan H. B. Wu (eds.)
Cardiac Markers is a complete, updated precis of scientific info on serum cardiac markers for coronary artery illness. This e-book reports intimately the biochemistry, scientific importance, and analytical measurements for every marker, in addition to the benefits and obstacles of present and proposed markers. finished descriptions of healing methods to regulate sufferers are awarded. additionally incorporated are up to date listings of fundamental references and advertisement methodologies, the most recent information at the pathophysiology of risky angina, and a dialogue of the recent emergency-room chest soreness facilities. a person who must know the way to check, diagnose, deal with, and deal with sufferers with acute chest discomfort and center assaults will locate this e-book indispensable.
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Extra resources for Cardiac Markers
Cardiovasc. Risk Factors 4:79-91. Fuster V, Badimon L, Badimon JJ, and Cheseboro JH (1992) The pathogensis of coronary artery disease and the acute coronary syndromes. N. Engl. J. Med. 326: 242-250,310-318. Astrup T (1956) The biological significance of fibrinolysis. Lancet 2:565-568. Jesperson J (1987) Pathophysiology and clinical aspects of fibrinolysis and inhibition of coagulation. Thesis, Laegeforeningens Forlag. Meade TW, Ruddock V, Stirling Y, Chakrabarti R, and Miller GJ (1993) Fibri- 37 20.
Further elucidation regarding the role of fibrinogen is needed because it is possible that raised plasma fibrinogen levels merely represent inflammation -induced acute-phase reaction (43). Factor VII activity has been shown to be independently associated with the risk of future CHD in middle-aged men (19). Also, several cross-sectional studies show increased factor VII mass and activity in patients with or at risk for CHD (43,44). A relationship between the dietary influence on plasma triglyceride concentration and phospholipid-factor VII complex has been postulated (45).
The normal endothelium helps modulate the vasomotor response of the vascular smooth muscle cells. Atherosclerosis impairs the normal vasodilator capability of the endothelium, and in the absence of normal endothelium, the smooth muscle cells will be constricted by thrombin, thromboxane A2 (TxA2), serotonin, and other agents. Thus far, a relationship between many of these factors and CHD remains hypothetical. Giri, Waters, and Wu 24 B OXIDIZED LDL • • • • • Chemotactic for Monocytes Chemostatic for Macropl1age Enhances Foam Cell Formation Causes Endotl1elial lnj ury In creased Production of Endot helin , .
Cardiac Markers by Alan H. B. Wu (auth.), Alan H. B. Wu (eds.)